首页/数据集/m6A independent genome-wide METTL3 and METTL14 redistribution drives senescence-associated secretory phenotype. m6A independent genome-wide METTL3 and METTL14 redistribution drives senescence-associated secretory phenotype

m6A independent genome-wide METTL3 and METTL14 redistribution drives senescence-associated secretory phenotype. m6A independent genome-wide METTL3 and METTL14 redistribution drives senescence-associated secretory phenotype

NIAID Data Ecosystem2026-03-11 收录

衰老相关分泌表型

m6A表观转录组学

数据链接：

https://www.ncbi.nlm.nih.gov/bioproject/PRJNA595517 数据链接链接失效反馈

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资源简介：

This SuperSeries is composed of the SubSeries listed below. Overall design: Refer to individual Series

应用场景：

创建时间：

2019-12-13

m6A表观转录组学

先天免疫调控

Full immunoblots images for cropped gels which appear in the paper.

NIAID Data Ecosystem70

The m6A methyltransferase Mettl3 deficiency attenuates hepatic stellate cell activation and liver fibrosis [m6A-RIP-seq]. The m6A methyltransferase Mettl3 deficiency attenuates hepatic stellate cell activation and liver fibrosis [m6A-RIP-seq]

肝纤维化

m6A表观转录组学

m6A-RIP sequencing of primary hepatic stellate cells (HSCs) isolated from Control and HSC-specific Mettl3-knockout (Mettl3 cKO) mouse liver tissues. Overall design: Control and Mettl3 cKO mouse primar

NIAID Data Ecosystem20

YTHDF3 improves translation initiation of mRNAs with N6-methyladenosine methylation at 5’ untranslated region by facilitating eIF2AK2 recruitment in oxaliplatin-resistant colorectal cancer - RIP-seq dataset. YTHDF3 improves translation initiation of mRNAs with N6-methyladenosine methylation at 5’ untranslated region by facilitating eIF2AK2 recruitment in oxaliplatin-resistant colorectal cancer - RIP-seq dataset

化疗耐药机制

m6A表观转录组学

Oxaliplatin as a first-line drug frequently causes the chemo-resistance on colorectal cancer (CRC). N6-methyladenosine (m6A) methylation has been largely acknowledged in multiple biological functions.

NIAID Data Ecosystem10

A novel RNA m6A modulator Zc3h13 plays an anchor role in facilitating nuclear RNA methylation and mouse embryonic stem cell self-renewal

m6A表观转录组学

胚胎干细胞自我更新

N6-methyladenosine (m6A) is an abundant modification in eukaryotic mRNA, regulating mRNA dynamics by influencing mRNA stability, splicing, export and translation. Recent studies discovered m6A methylt

NIAID Data Ecosystem20

Inhibition of METTL3 attenuates renal injury and inflammation by alleviating TAB3 m6A modifications via IGF2BP2-dependent mechanisms

脓毒症急性器官损伤分子机制

m6A表观转录组学

The role of N6-methyladenosine (m6A) modifications in renal diseases is largely unknown. Here, we characterised the role of N6-adenosine-methyltransferase METTL3, whose expression is elevated in renal

NIAID Data Ecosystem10

m6A independent genome-wide METTL3 and METTL14 redistribution drives senescence-associated secretory phenotype. m6A independent genome-wide METTL3 and METTL14 redistribution drives senescence-associated secretory phenotype

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