Expression in LT-HSC after in vitro culture in mSCF, mTpo, mFlt3L, hIGFBP2 and Angptl5.

ChEBI2011-12-28 更新2026-07-21 收录

造血干细胞培养

细胞因子调控

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Expression in LT-HSC after in vitro culture in mSCF, mTpo, mFlt3L, hIGFBP2 and Angptl5.

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2011-12-28

Caspase-6 mediates resistance against Burkholderia pseudomallei infection and influences the expression of detrimental cytokines

感染免疫机制

细胞因子调控

Caspase-6 is a member of the executioner caspases and known to play a role in innate and adaptive immune processes. However, its role in infectious diseases has rarely been addressed yet. We here exam

NIAID Data Ecosystem90

Immunomodulatory activities of peptides IDR-HH2, IDR-1002 and IDR 1018 in human peripheral blood mononuclear cells (PBMC).

免疫调节肽

细胞因子调控

Cells were stimulated with 20 µg/ml of peptide. 1Experiments were performed 2–4 times and means are presented. All values for peptide treated cells represent significant induction relative to the untr

NIAID Data Ecosystem60

Polyvinyl alcohol expanded mouse HSC cultures (bulk RNA-Seq)

造血干细胞培养

转录组学分析

We previously reported a polyvinyl alcohol-based mouse hematopoietic stem cell (HSC) culture protocol that efficiently expanded transplantable HSCs for at least a month ex vivo (Wilkinson et al., Natu

NIAID Data Ecosystem50

GO:0032736 (positive regulation of interleukin-13 production)

细胞因子调控

生物通路挖掘

This network was assembled automatically by INDRA (http://indra.bio) by processing all available biomedical literature with multiple machine reading systems, and integrating curated pathway databases.

NIAID Data Ecosystem60

Interleukin-6 Deficiency Influences Cytokine Expression in Susceptible BALB Mice Infected with Leishmania major but Does Not Alter the Outcome of Disease

感染免疫学

细胞因子调控

Since interleukin-6 (IL-6) may promote Th2 responses, we infected BALB IL-6-deficient (IL-6(−/−)) mice with Leishmania major. There was not a significant difference between the courses of infection (l

PubMed Central50

Expression in LT-HSC after in vitro culture in mSCF, mTpo, mFlt3L, hIGFBP2 and Angptl5.

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