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Cell lines treated with BrdU against DMSO controls (DNA damage-induced senescence)

ChEBI2025-10-15 更新2026-07-12 收录

细胞衰老机制

DNA损伤响应

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-MTAB-15005 数据链接链接失效反馈

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Cell lines treated with BrdU against DMSO controls (DNA damage-induced senescence)

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创建时间：

2025-10-15

细胞衰老机制

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辐射表观遗传学

细胞衰老机制

The project is aimed at the analysis of epigenetic effects of cellular responses to irradiation. Here we aim to sequence genomes, epigenomes, and transcriptomes of cells exposed to different kinds of

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DNA damage primes hematopoietic stem cells for megakaryopoiesis. DNA damage primes hematopoietic stem cells for megakaryopoiesis

造血干细胞

DNA损伤响应

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Translational and transcriptional responses of fission yeast to MMS

DNA损伤响应

裂殖酵母基因表达

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ChEBI2020-11-18 更新40

Chk2 is dispensable for p53-mediated G(1) arrest but is required for a latent p53-mediated apoptotic response

DNA损伤响应

细胞周期检查点

In response to genotoxic stress, mammalian cells can activate cell cycle checkpoint pathways to arrest the cell for repair of DNA damage or induce apoptosis to eliminate damaged cells. The checkpoint

PubMed Central2002-07-03 更新10

Cell lines treated with BrdU against DMSO controls (DNA damage-induced senescence)

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