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Growth hormone releasing hormone signaling promotes Th17 cell differentiation and autoimmune inflammation

NIAID Data Ecosystem2026-05-01 收录

数据链接：

https://figshare.com/articles/dataset/Growth_hormone_releasing_hormone_signaling_promotes_Th17_cell_differentiation_and_autoimmune_inflammation/22739690 数据链接链接失效反馈

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The file contains all raw data presented in all Figures.

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创建时间：

2023-06-07

Th17细胞

Il24基因功能

NaÃ¯ve T cells (CD3e+, CD4+, CD44-, CD62L+, CD25-) were isolated from splenocytes and lymph nodes of Il24 â/â mice (B6N(Cg)-Il24tm1.1(KOMP)Vlcg/2J) and Il24 +/+ wild type littermates (C57BL/6) usi

NIAID Data Ecosystem110

total RNA-seq from T-helper 17 cell (ENCSR831GLL)

Th17细胞

单细胞RNA测序

h.CD4.Th17_DONOR 6: 0 hour no activation For data usage terms and conditions, please refer to http://www.genome.gov/27528022 and http://www.genome.gov/Pages/Research/ENCODE/ENCODE_Data_Use_Policy_for

NIAID Data Ecosystem90

Analysis of transcriptomes of naive CD4+ T cells, polarized under Th17 cell skewing conditions for 24h in presence or abscence of CK2 Inhibitor. Mus musculus

Th17细胞

转录组学分析

The goal of this study was to elucidate the transcriptional changes evoked by CK2 inhibition in developing Th17 cells Overall design: Messenger RNA profiles of naive CD4+ T cells, FACS-purifed from sp

NIAID Data Ecosystem40

Pluripotent stem cell models of Blau syndrome reveal an IFN- -dependent inflammatory response in macrophages

干细胞研究

自身炎症疾病

Background: Blau syndrome, or early-onset sarcoidosis, is a juvenile-onset systemic granulomatosis associated with a mutation in Nucleotide-binding oligomerization domain 2 (NOD2). The underlying mech

干细胞与再生医学数据中心2022-02-20 更新100

Global gene expression profile of wild type and Smad4-deficient Th17 cells cultured at 37°C and 39.5°C

Th17细胞

发热温度免疫调控

Febrile temperature enhanced Th17 differentiation in Smad4 SUMOylation-dependent manner. Smad4-deficiency impaired the febrile temperature-enhanced Th17 differentiation. In order to understand how feb

NIAID Data Ecosystem90

Growth hormone releasing hormone signaling promotes Th17 cell differentiation and autoimmune inflammation

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